Dysregulation of PRMT5 in chronic lymphocytic leukemia promotes progression with high risk of Richter's transformation.

TitleDysregulation of PRMT5 in chronic lymphocytic leukemia promotes progression with high risk of Richter's transformation.
Publication TypeJournal Article
Year of Publication2023
AuthorsHing ZA, Walker JS, Whipp EC, Brinton L, Cannon M, Zhang P, Sher S, Cempre CB, Brown F, Smith PL, Agostinelli C, Pileri SA, Skinner JN, Williams K, Phillips H, Shaffer J, Beaver LP, Pan A, Shin K, Gregory CT, Ozer GH, Yilmaz SA, Harrington BK, Lehman AM, Yu L, Coppola V, Yan P, Scherle P, Wang M, Pitis P, Xu C, Vaddi K, Chen-Kiang S, Woyach J, Blachly JS, Alinari L, Yang Y, Byrd JC, Baiocchi RA, Blaser BW, Lapalombella R
JournalNat Commun
Volume14
Issue1
Pagination97
Date Published2023 Jan 06
ISSN2041-1723
KeywordsAnimals, Leukemia, Lymphocytic, Chronic, B-Cell, Lymphoma, Large B-Cell, Diffuse, Mice
Abstract

Richter's Transformation (RT) is a poorly understood and fatal progression of chronic lymphocytic leukemia (CLL) manifesting histologically as diffuse large B-cell lymphoma. Protein arginine methyltransferase 5 (PRMT5) is implicated in lymphomagenesis, but its role in CLL or RT progression is unknown. We demonstrate herein that tumors uniformly overexpress PRMT5 in patients with progression to RT. Furthermore, mice with B-specific overexpression of hPRMT5 develop a B-lymphoid expansion with increased risk of death, and Eµ-PRMT5/TCL1 double transgenic mice develop a highly aggressive disease with transformation that histologically resembles RT; where large-scale transcriptional profiling identifies oncogenic pathways mediating PRMT5-driven disease progression. Lastly, we report the development of a SAM-competitive PRMT5 inhibitor, PRT382, with exclusive selectivity and optimal in vitro and in vivo activity compared to available PRMT5 inhibitors. Taken together, the discovery that PRMT5 drives oncogenic pathways promoting RT provides a compelling rationale for clinical investigation of PRMT5 inhibitors such as PRT382 in aggressive CLL/RT cases.

DOI10.1038/s41467-022-35778-1
Alternate JournalNat Commun
PubMed ID36609611
PubMed Central IDPMC9823097
Grant ListTL1 TR002735 / TR / NCATS NIH HHS / United States
R01 CA260858 / CA / NCI NIH HHS / United States
R01 CA177292 / CA / NCI NIH HHS / United States
R01 CA240493 / CA / NCI NIH HHS / United States
R01 CA193167 / CA / NCI NIH HHS / United States
P30 CA016058 / CA / NCI NIH HHS / United States
P01 CA214274 / CA / NCI NIH HHS / United States
T32 GM068412 / GM / NIGMS NIH HHS / United States
R01 CA214046 / CA / NCI NIH HHS / United States
R35 CA197734 / CA / NCI NIH HHS / United States
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