Pathology & Laboratory Medicine

Publications

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2018

Magro CM, Olson LC. Small cell lymphocytic variant of marginal zone lymphoma: A distinct form of marginal zone lymphoma derived from naïve B cells as a cutaneous counterpart to the naïve marginal zone lymphoma of splenic origin. Ann Diagn Pathol. 2018;34:116-121.

Ooki A, Pena MD, Marchionni L, Dinalankara W, Begum A, Hahn NM, et al. YAP1 and COX2 Coordinately Regulate Urothelial Cancer Stem-like Cells. Cancer Res. 2018;78(1):168-181.

Cushing MM, Kelley J, Klapper E, Friedman DF, Goel R, Heddle NM, et al. Critical developments of 2017: a review of the literature from selected topics in transfusion. A committee report from the AABB Clinical Transfusion Medicine Committee. Transfusion. 2018;58(4):1065-1075.

Huang J, Duran A, Reina-Campos M, Valencia T, Castilla EA, Müller TD, et al. Adipocyte p62/SQSTM1 Suppresses Tumorigenesis through Opposite Regulations of Metabolism in Adipose Tissue and Tumor. Cancer Cell. 2018;33(4):770-784.e6.

Scognamiglio T, Chen Y-. Beyond the Percentages of PD-L1-Positive Tumor Cells: Induced Versus Constitutive PD-L1 Expression in Primary and Metastatic Head and Neck Squamous Cell Carcinoma. Head Neck Pathol. 2018;12(2):221-229.

Alves CP, Dey-Guha I, Kabraji S, Yeh AC, Talele NP, Solé X, et al. AKT1 Quiescent Cancer Cells Promote Solid Tumor Growth. Mol Cancer Ther. 2018;17(1):254-263.

Arenas MA, Whitsett TG, Aronova A, Henderson SA, LoBello J, Habra MA, et al. Protein Expression of PTTG1 as a Diagnostic Biomarker in Adrenocortical Carcinoma. Ann Surg Oncol. 2018;25(3):801-807.

Liu D, Takhar M, Alshalalfa M, Erho N, Shoag J, Jenkins RB, et al. Impact of the SPOP Mutant Subtype on the Interpretation of Clinical Parameters in Prostate Cancer. JCO Precis Oncol. 2018;2018.

Goyal A, Zhang G, Yang B. Differential expression patterns of GATA3 in usual and differentiated types of vulvar intraepithelial neoplasia: potential diagnostic implications. Mod Pathol. 2018;31(7):1131-1140.

Ko H-, Berk M, Chung Y-, Willard B, Bareja R, Rubin M, et al. Loss of an Androgen-Inactivating and Isoform-Specific HSD17B4 Splice Form Enables Emergence of Castration-Resistant Prostate Cancer. Cell Rep. 2018;22(3):809-819.