Pathology & Laboratory Medicine

Publications

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2018

Trindade AJ, Fan C, Cheung M, Greenberg RE. Endoscopic resection of an esophageal schwannoma. Dig Liver Dis. 2018;50(3):309.

Magro CM, Olson LC. Small cell lymphocytic variant of marginal zone lymphoma: A distinct form of marginal zone lymphoma derived from naïve B cells as a cutaneous counterpart to the naïve marginal zone lymphoma of splenic origin. Ann Diagn Pathol. 2018;34:116-121.

Hung PJ, Johnson B, Chen B-, Byrum AK, Bredemeyer AL, Yewdell WT, et al. MRI Is a DNA Damage Response Adaptor during Classical Non-homologous End Joining. Mol Cell. 2018;71(2):332-342.e8.

Chiaretti S, Messina M, Grammatico S, Piciocchi A, Fedullo AL, Di Giacomo F, et al. Rapid identification of BCR/ABL1-like acute lymphoblastic leukaemia patients using a predictive statistical model based on quantitative real time-polymerase chain reaction: clinical, prognostic and therapeutic implications. Br J Haematol. 2018;181(5):642-652.

Scognamiglio T, Chen Y-. Beyond the Percentages of PD-L1-Positive Tumor Cells: Induced Versus Constitutive PD-L1 Expression in Primary and Metastatic Head and Neck Squamous Cell Carcinoma. Head Neck Pathol. 2018;12(2):221-229.

Ng SY, Yoshida N, Christie AL, Ghandi M, Dharia NV, Dempster J, et al. Targetable vulnerabilities in T- and NK-cell lymphomas identified through preclinical models. Nat Commun. 2018;9(1):2024.

Alves CP, Dey-Guha I, Kabraji S, Yeh AC, Talele NP, Solé X, et al. AKT1 Quiescent Cancer Cells Promote Solid Tumor Growth. Mol Cancer Ther. 2018;17(1):254-263.

Liu D, Takhar M, Alshalalfa M, Erho N, Shoag J, Jenkins RB, et al. Impact of the SPOP Mutant Subtype on the Interpretation of Clinical Parameters in Prostate Cancer. JCO Precis Oncol. 2018;2018.

Goyal A, Zhang G, Yang B. Differential expression patterns of GATA3 in usual and differentiated types of vulvar intraepithelial neoplasia: potential diagnostic implications. Mod Pathol. 2018;31(7):1131-1140.

Ko H-, Berk M, Chung Y-, Willard B, Bareja R, Rubin M, et al. Loss of an Androgen-Inactivating and Isoform-Specific HSD17B4 Splice Form Enables Emergence of Castration-Resistant Prostate Cancer. Cell Rep. 2018;22(3):809-819.